Carbohydrates, Friend or Foe?

Learn how carbs, starches, and sugars affect metabolism — and why bread can raise blood sugar as fast as soda.

Jay Simons
November 9, 2025

What Are Carbohydrates?

Carbohydrate is a broad term. They come in several forms. The scientific definition is:

An organic molecule composed of carbon (C), hydrogen (H), and oxygen (O), typically with the general formula (CH₂O)ₙ, where n is three or greater.

That definition covers a lot of different compounds! Here are the three basic categories of dietary carbs:

  • Simple carbohydrates: single or double sugar molecules (monosaccharides and disaccharides) like glucose, fructose, and sucrose.
  • Complex carbohydrates: long chains of sugars called polysaccharides, such as starch and fiber.
  • Engineered carbohydrates: synthetic sugars like maltodextrin, dextrose, and high-fructose corn syrup (HFCS), which are absorbed even faster than table sugar.

All carbohydrates ultimately become glucose in the bloodstream. The difference lies in how quickly that happens. And that’s the key.


The Body’s Real Relationship With Carbohydrates

While glucose is vital for energy, the body does not need to eat carbohydrates to make it.
Through a process called gluconeogenesis (“creating new glucose”), the liver and kidneys can produce glucose from amino acids, glycerol (fat), and protein, in that order. Your body will even digest its own muscle tissue if you go without food long enough.

In other words, carbohydrates are non-essential nutrients. The body can make all the glucose it needs without them.

However, modern diets supply far more than needed, and the excess becomes a metabolic burden.


Starch and Sugar: Two Sides of the Same Coin

Starch is essentially stored sugar. It is a long chain of glucose molecules (polysaccharides) that your body breaks down into glucose during digestion. Enzymes like amylase and maltase chop starch into smaller sugars that absorb quickly into the blood.

A green banana is a perfect example: when unripe, it tastes starchy because it contains resistant starch (which digests slowly). As it ripens, enzymes convert that starch into simple sugars, making it sweet. Bread, pasta, and cereals work the same way; they start as starch but end up as sugar in your bloodstream.

Even store-bought “whole grain” bread is not much better. Milling destroys the grain’s natural fiber structure, so your body absorbs it almost as fast as pure sugar.
According to international glycemic index (GI) tables, most commercial breads — white, wheat, or “multigrain” — have a GI around 70–75, nearly identical to table sugar (≈65) and a can of sugary soda (≈70–75).
In other words, a slice of bread can raise your blood sugar about as fast as soda.


The Insulin Overshoot Cycle

When glucose enters your blood, your pancreas releases insulin to move it into your cells for energy or storage.
Highly processed carbs and sugary foods cause a sharp insulin spike, followed by a steep drop in blood sugar (reactive hypoglycemia).
This “crash” makes you hungry and tired, leading you to reach for more sugar.
Your pancreas “remembers” this overreaction (insulin secretion priming), releasing even more insulin next time. Over time, your cells stop responding to insulin, a state called insulin resistance.

As insulin resistance worsens, glucose remains trapped in the bloodstream while your cells are starved for energy. Doctors often recommend “eating sugar” to fix low blood sugar, restarting the same destructive cycle. The disease they call Type 2 diabetes is nothing more than advanced insulin resistance.


How Chronic High Blood Sugar Damages the Body

When blood glucose remains high for long periods, sugar molecules stick to proteins and fats in the body, a process called glycation. This produces advanced glycation end products (AGEs) that cause inflammation and damage to tissues.
Over time, this leads to:

  • Cardiovascular disease: stiff arteries and plaque buildup
  • Neuropathy: nerve damage and chronic pain
  • Nephropathy: kidney damage
  • Retinopathy: blindness from damaged blood vessels in the eyes
  • Cognitive decline: insulin resistance in the brain, sometimes called “type 3 diabetes”

These changes develop silently over years and often become permanent. Many people with advanced, untreated cases of diabetes, completely or partially lose their vision, and even lose legs due to infection.


Sugar, Alcohol, and the Body’s Fuel Order

Sugar and alcohol are metabolically linked. Yeast ferments sugar into ethanol (alcohol), and in the body, both are handled by the liver.
Your body burns energy in this order:

  1. Alcohol: burned first because it is toxic; the liver must detoxify it before anything else.
  2. Carbohydrates (glucose and glycogen): used next for immediate energy.
  3. Fat: burned last, only when the other two are gone.

That is why regular alcohol consumption halts fat burning. As long as alcohol or sugar is in the system, your body will not efficiently burn fat. In fact, even if you lower your carbohydrate intake, it takes weeks to start burning your stored fat because your ketone (metabolic fuels) levels will be very low because your liver produces them when you burn mostly fat for energy. It takes time for the liver to produce these them efficiently.


Alcoholism and Diabetes

Chronic alcohol use damages the pancreas and liver, the two organs most responsible for blood sugar control.

Alcohol depletes stored glucose (glycogen), increases insulin resistance, and promotes fatty liver disease.

Alcoholism and diabetes feed off each other: poor blood sugar control worsens alcohol’s toxic effects, while heavy drinking makes diabetes harder to manage. Even moderate drinking can raise triglycerides and disrupt insulin sensitivity in people already at risk.

It is absolutely imperative that anyone with Type 2 diabetes do not drink!


The Healthier Carbohydrate Spectrum

Not all carbohydrates are harmful. The key is how fast and in what form they enter your bloodstream.

Whole, unprocessed foods — fruits, vegetables, beans, and foods high in fiber or resistant starch — slow digestion and stabilize blood sugar.

Resistant starch (found in lentils, unripe bananas, and cooled potatoes) acts like fiber, feeding gut bacteria and improving insulin sensitivity.


What the Heck is the Glycemic Index?

The glycemic index (GI) is a scientific scale that measures how quickly a carbohydrate-containing food raises blood glucose levels compared to pure glucose (blood sugar), which has a GI value of 100. Foods with a high GI (70 or above) are digested and absorbed rapidly, causing sharp spikes in blood sugar and insulin response, while low-GI foods (55 or below) break down more slowly, leading to a gradual, sustained release of energy. The GI reflects not just the type of carbohydrate, but also factors like processing, fiber content, and how the food is cooked. It is an important tool for understanding how different foods affect metabolism, appetite, and long-term risks of insulin resistance and diabetes.

Glycemic Index (GI) and Glycemic Load (GL) Chart

Food ItemCategoryGI ValueGL (per serving)Notes
Glucose (reference)Sugar10010 (10 g)Baseline standard
White bread (1 slice)Refined starch7510Rapid glucose spike
Whole wheat bread (1 slice)Refined starch749Nearly identical to white
Multigrain bread (1 slice)Refined starch728Often contains added sugars
Bagel (½ medium)Refined starch7217Extremely high load
Cornflakes (1 cup)Breakfast cereal8120High GI and GL
Oatmeal (rolled oats, 1 cup cooked)Whole grain5513Lower if steel-cut
Rice, white (1 cup cooked)Refined starch7323Common high-carb staple
Rice, brown (1 cup cooked)Whole grain6818More fiber, slightly lower GL
Potato, baked (1 medium, russet)Starch (root)7826One of the highest
Sweet potato (1 medium, boiled)Starch (root)6317Lower when cooled
Banana (ripe, 1 medium)Fruit (simple sugar)5113GI rises as it ripens
Banana (green/unripe)Resistant starch306Digests slowly
Apple (1 medium)Fruit386Fructose slows absorption
Orange (1 medium)Fruit405Low GI, high fiber
Watermelon (1 cup)Fruit724High GI, but low load due to water content
Carrots (boiled, 1 cup)Vegetable393Minimal glucose impact
Beans (kidney, ½ cup)Legume297High fiber and resistant starch
Lentils (boiled, ½ cup)Legume325Excellent for blood sugar control
Milk (whole, 1 cup)Dairy414Lactose digested slowly
Yogurt (plain, unsweetened, ¾ cup)Dairy353Probiotics help digestion
Sucrose (table sugar, 1 tbsp)Sugar657Glucose + fructose mix
Honey (1 tbsp)Sugar619Variable by flower source
Soda (Mountain Dew, 12 oz)Sweet beverage7116Rapid blood sugar spike
Orange juice (8 oz)Sweet beverage6613Less fiber than whole fruit
Peanuts (1 oz)Fat/protein source141Minimal effect
Ice cream (½ cup)Sugar + fat combo578Fat slows absorption
Dark chocolate (70% cocoa, 1 oz)Fat + fiber combo232Low GI, rich in antioxidants

GI and GL Classification

CategoryGI RangeGL Range (per serving)Meaning
Low55 or less10 or lessSlow glucose release; minimal blood sugar impact
Medium56–6911–19Moderate rise; controlled insulin response
High70+20+Rapid spike; strong insulin response

What Does All This Mean?

  • GI measures how fast a food raises blood sugar.
  • GL measures how much a normal serving affects blood sugar.
  • Foods like watermelon have a high GI but low GL, meaning they digest quickly but don’t contain much total sugar.
  • Foods like white rice or potatoes are high in both, creating large and rapid spikes.
  • Combining carbs with fat, fiber, or protein slows absorption and lowers both GI and GL impact.

TL;DR

Starches, sugars, and alcohol belong to the same metabolic family, quick-burning fuels that are safe in small amounts but harmful in excess.
The real danger lies not in carbohydrates themselves but in their speed, form, and frequency.
The human body does not need to eat carbohydrates to maintain glucose; it can make all it requires from protein and fat.
Modern processed foods, breads, and sweetened drinks cause constant insulin surges that wear down your metabolism.
The path to better health is simple:

  • Limit refined carbs and alcohol,
  • Emphasize whole, fibrous foods,
  • Let your body burn fat, not just sugar.

Sources

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